In humans, acupuncture at ST36 was found to improve gastric dysrhythmias and abdominal symptoms induced by RD. In this study, as expected, EA at ST36 normalized or prevented RD-induced gastric dysrhythmias. Interestingly, AEA showed a similar normalizing or preventive Tubacin customer reviews effect on RD-induced gastric dysrhythmias. The percentage of normal GSW during RD was 94% that was almost the same as that before RD. Further analysis revealed complete elimination of tachygastria and almost complete elimination of arrhythmia with AEA during RD. The ameliorating effect of AEA on RD-induced gastric dysrhythmias is believed to be mediated via the vagal mechanism. Anatomically, the gastric point in the ear is innervated with Arnold nerve, a branch of the vagus. It was shown that needling gastric points in auricular of rats caused the expression of neuropeptide Y in the hypothalamus increased significantly in the fed state and some feeding-related hypothalamic neuronal activities were modulated significantly after stimulating the cavum conchae areas in auriculars of obese rats with low frequency electroacupuncture. In the current study, the effect of AEA on gastric dysrhythmias was completely blocked by atropine, a muscarinic receptor antagonist known to block vagal nerve activity. These findings suggested a vago-vagal mechanism involved in the ameliorating effect of AEA on RD-induced gastric dysrhythmia; electrical stimulation at the gastric point in the ear activated the vagal nerve that led to a central response in the brain stem, such as dorsal motor nucleus of the vagus or in the hypothalamus, and the activation of the central nerves by AEA solicited an enhanced vagal efferent activity to the stomach, resulting in improvement in gastric slow waves. The similar vagal mechanism was also consistently reported with EA at ST36 in our previous studies. In both humans and dogs, RD induced gastric dysrhythmias and reduced vagal activity measured by the spectral analysis of the heart rate variability; EA in dogs or acupuncture in humans at ST36 increased vagal activity and improved gastric slow waves during RD. In diabetic rats, EA at ST36 was also reported to improve gastric motility of model rats via vagal pathways. The other two major parameters, dominant frequency and dominant power of gastric slow wave recordings were not significantly altered by RD. It should be pointed out. However, these two parameters reflect the averages of the entire recording, unlike the percentage of normal slow waves or percentage of tachygastria/ bradygastria/ arrhythmia that reflect minuteby-minute changes in slow wave rhythmicity. It is conceivable that the overall parameters were less sensitive to intervention. It is well known that ST36 and PC6 are good acupoints in regulating gastrointestinal functions not only in humans but also in animals. However, up to now, there has not been any report comparing the long term effects of AEA and body electroacupuncture at ST36 or P6 on gastrointestinal motility in humans. Further studies are needed to determine whether AEA is better than EA at ST36 or PC6, or vice versa. Hyperuricemia is a metabolic problem that has become increasingly common worldwide.